@misc{Kapuściński_Andrzej_(1937–2018)_Is_1981,
 author={Kapuściński, Andrzej (1937–2018)},
 editor={Pluta, Ryszard},
 editor={Mossakowski, Mirosław J. (1929–2001)},
 copyright={Creative Commons Attribution BY 4.0 license},
 address={New York},
 howpublished={online},
 year={1981},
 publisher={Raven Press},
 language={eng},
 abstract={Systemic arterial hypotension is a well-known factor limiting recovery from cerebral ischemia. The majority of studies have mentioned its noxious effect on reperfusion of cerebral microcirculation and have dealt with methods of counteracting it rather than with its mechanism. Several authors assume an impairment of the vasomotor centers in midbrain and hypothalamus as a basie event in the mechanism of hypotension (1,2,9). The necessity of pharmacological support of blood pressure during the hypotensive period in the extensive research on complete cerebral ischemia by means of several experimental models turned our attention to the possible role of the lack of endogenic vasopressor substances, among other reasons. The purpose of this study was to characterize alterations of systemie arterial pressure during and after complete cerebral ischemia. and to correlate them with changes of noradrenaline concentration in the blood, as well as to analyze other factors participating in the mechanisms of ischemic-postischemic hypotension.},
 title={Is Acute failure of Adrenals an Important Factor Limiting Recovery from cerebral Ischemia ?},
 type={Text},
 URL={http://rcin.org.pl/Content/69383/PDF/Is%20Acute%20Failure.pdf},
 keywords={Cerebral Ischemia, Cerebral Microcirculation},
}